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Mol. Cells 2011; 31(2): 199-199

Published online February 15, 2011

https://doi.org/10.1007/s10059-011-0058-y

© The Korean Society for Molecular and Cellular Biology

Erratum to: The Neural Stem Cell Fate Determinant TLX Promotes Tumorigenesis and Genesis of Cells Resembling Glioma Stem Cells

Hyo-Jung Park, Jun-Kyum Kim, Hye-Min Jeon, Se-Yeong Oh, Sung-Hak Kim, Do-Hyun Nam1, and Hyunggee Kim*

Cell Growth Regulation Laboratory, School of Life Sciences and Biotechnology, Korea University, Seoul 136-713, Korea, 1Department of Neurosurgery, Samsung Medical Center and Samsung Biomedical Research Institute, Sungkyunkwan University School of Medicine, Seoul 135-710, Korea

Correspondence to : *Correspondence: hg-kim@korea.ac.kr

Abstract

This paper was initially submitted to Journal ‘Molecules and Cells’ without approval from two coauthors. According to the decision of editorial committee, I, as a corresponding author for this paper, withdraw authorship of two coauthors who did not agree to use of XO1, XO2, and XO3 cells shown in Fig. 1B. Thus, we change data shown in Fig. 1B by removing expression of TLX in these cells. We deeply regret this circumstance and apologize to the community.

Article

Erratum

Mol. Cells 2011; 31(2): 199-199

Published online February 28, 2011 https://doi.org/10.1007/s10059-011-0058-y

Copyright © The Korean Society for Molecular and Cellular Biology.

Erratum to: The Neural Stem Cell Fate Determinant TLX Promotes Tumorigenesis and Genesis of Cells Resembling Glioma Stem Cells

Hyo-Jung Park, Jun-Kyum Kim, Hye-Min Jeon, Se-Yeong Oh, Sung-Hak Kim, Do-Hyun Nam1, and Hyunggee Kim*

Cell Growth Regulation Laboratory, School of Life Sciences and Biotechnology, Korea University, Seoul 136-713, Korea, 1Department of Neurosurgery, Samsung Medical Center and Samsung Biomedical Research Institute, Sungkyunkwan University School of Medicine, Seoul 135-710, Korea

Correspondence to:*Correspondence: hg-kim@korea.ac.kr

Abstract

This paper was initially submitted to Journal ‘Molecules and Cells’ without approval from two coauthors. According to the decision of editorial committee, I, as a corresponding author for this paper, withdraw authorship of two coauthors who did not agree to use of XO1, XO2, and XO3 cells shown in Fig. 1B. Thus, we change data shown in Fig. 1B by removing expression of TLX in these cells. We deeply regret this circumstance and apologize to the community.

Mol. Cells
Sep 30, 2023 Vol.46 No.9, pp. 527~572
COVER PICTURE
Chronic obstructive pulmonary disease (COPD) is marked by airspace enlargement (emphysema) and small airway fibrosis, leading to airflow obstruction and eventual respiratory failure. Shown is a microphotograph of hematoxylin and eosin (H&E)-stained histological sections of the enlarged alveoli as an indicator of emphysema. Piao et al. (pp. 558-572) demonstrate that recombinant human hyaluronan and proteoglycan link protein 1 (rhHAPLN1) significantly reduces the extended airspaces of the emphysematous alveoli by increasing the levels of TGF-β receptor I and SIRT1/6, as a previously unrecognized mechanism in human alveolar epithelial cells, and consequently mitigates COPD.

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